HomeLearning HubIB DP BiologyD2.2 Gene expression
D2.2

Gene expression

Theme D · Continuity and change · Cells · Additional higher level only

This entire topic is additional higher level. SL candidates are not examined on any of it. Every cell in your body carries the same genome, yet a neuron and a liver cell could hardly be more different. The difference is not in which genes they have but in which genes they use. This topic is about how gene expression is switched on and off, how those settings can be copied into daughter cells and even offspring without any change to the DNA sequence, and how the environment can reset them.

🎯What you need to be able to do

  • Explain gene expression as the mechanism by which genes affect phenotype.
  • Explain regulation of transcription by promoters, enhancers and transcription factors.
  • Explain control of mRNA degradation as a way of regulating translation.
  • Define epigenesis, and distinguish the genome, transcriptome and proteome.
  • Explain methylation of promoters and of histones as epigenetic tags.
  • Explain epigenetic inheritance, and outline environmental effects on gene expression, including air pollution.
  • Explain the consequences of removing most epigenetic tags from gametes, using tigons and ligers.
  • Explain how monozygotic twin studies investigate environmental effects on gene expression.
  • Give examples of a hormone and of a biochemical (lactose in bacteria) altering the pattern of gene expression.

📚The biology

Gene expression and phenotype

Gene expression is the mechanism by which the information in a gene has an effect on the phenotype. The most common route has three stages:

  1. Transcription of the gene into mRNA (D1.2);
  2. Translation of the mRNA into a polypeptide;
  3. The function of the protein product — for example an enzyme that catalyses a reaction producing a pigment, which gives a visible trait.

Expression can be controlled at each stage, so there are many points at which a cell can decide how much of a protein to make.

Regulating transcription

Transcription is controlled by proteins that bind to specific base sequences in DNA:

Promoter
A sequence immediately before a gene, where RNA polymerase and general transcription factors bind to start transcription.
Enhancers
Sequences that can be some distance from the gene. When specific activator proteins bind to them, the DNA loops round so they interact with the promoter complex, increasing the rate of transcription. (Similar sequences called silencers bind repressors and reduce it.)
Transcription factors
Proteins that bind to promoters or enhancers and help or block the binding of RNA polymerase. Which transcription factors a cell makes, and whether they are active, determines which genes it transcribes.

mRNA degradation

Once mRNA is made, the amount of protein produced depends on how long the mRNA survives in the cytoplasm before it is broken down by enzymes called nucleases. In human cells, mRNA may persist for anything from minutes to days. Controlling the rate of degradation is another way of regulating translation: mRNA for proteins needed only briefly, such as signalling proteins, is broken down quickly; mRNA for proteins needed constantly lasts longer.

Epigenesis

Epigenesis is the development of patterns of differentiation in the cells of a multicellular organism: as an embryo develops, cells take on different, stable patterns of gene expression and become different cell types. The key point is that DNA base sequences are not altered by epigenetic changes: the phenotype of the cell changes, but its genotype does not.

Genome, transcriptome and proteome

Genome
All the genetic information of an organism — the complete DNA sequence. The same in almost every cell of the body.
Transcriptome
All the RNA molecules transcribed in a cell at a particular time. Differs between cell types and changes over time.
Proteome
All the proteins produced by a cell (or organism) at a particular time. Differs between cells and changes with conditions.

No cell expresses all of its genes. The pattern of gene expression in a cell — its transcriptome and proteome — determines how it differentiates.

Epigenetic tags: methylation

Chemical groups attached to DNA or to histones act as epigenetic tags, marking genes to be expressed or silenced. Two examples:

  • Methylation of the promoter. Methyl groups (–CH3) can be added to cytosine bases in DNA. When the promoter of a gene is heavily methylated, transcription factors and RNA polymerase cannot bind, so transcription is repressed and the gene downstream is not expressed.
  • Methylation of histones. Methyl groups can also be added to certain amino acids in the histone proteins of nucleosomes. Depending on which amino acid is methylated, this can make the DNA pack more tightly (repressing transcription) or more loosely (activating it). (Details are not required.)

Epigenetic inheritance

Epigenetic tags can be copied when a cell divides. After DNA replication, enzymes recognize methylation on the old strand and add methyl groups to the matching positions on the new strand. Tags that remain in place during mitosis are passed to daughter cells, which is how a liver cell’s descendants stay liver cells. Occasionally, tags remain in place during meiosis and are passed to offspring. In both cases, a phenotypic change is inherited without any change to the nucleotide sequence of DNA.

Environmental effects on gene expression

The environment can change epigenetic tags and so alter gene expression. For example, exposure to air pollution, such as fine particulate matter from traffic, has been found to alter the pattern of methyl tags on DNA in blood cells, including at genes involved in inflammation and the response to oxidative stress. This may help to explain how pollution increases the risk of respiratory and cardiovascular disease. Diet, smoking, stress and temperature are other environmental factors shown to affect methylation.

Resetting tags in gametes: tigons and ligers

When sperm and eggs form, most, but not all, epigenetic tags are removed, so that the zygote can start development with a largely fresh pattern. The tags that remain can have striking consequences.

Some genes are imprinted: the copy from one parent is silenced by tags that survive into the embryo, so only the copy from the other parent is expressed. Some imprinted genes affect growth, with the father’s copies tending to promote growth of the offspring and the mother’s copies tending to restrain it.

  • A liger (lion father × tiger mother) grows much larger than either parent. In lions, where females mate with several males, paternal growth-promoting genes are strongly expressed and maternal genes have evolved to counteract them. A tiger mother does not have that counterbalance, so the lion father’s growth-promoting imprinting goes unchecked.
  • A tigon (tiger father × lion mother) is similar in size or smaller than its parents, because the lion mother’s growth-restraining imprinting is now combined with the tiger father’s weaker growth promotion.

Both hybrids receive the same two sets of genes. The difference in phenotype depends on which parent each set came from — an epigenetic, not a genetic, effect.

Monozygotic twin studies

Monozygotic (identical) twins develop from one zygote, so they have identical genomes. Any differences between them in phenotype must therefore come from the environment, including its effects on gene expression. Studies comparing twins have found that their patterns of DNA methylation are very similar at birth but become increasingly different with age, and more different in twins who have lived apart or had different lifestyles. This is evidence that the environment changes gene expression over a lifetime.

External factors that change gene expression

A hormone: oestradiol
Oestradiol diffuses into target cells and binds to an intracellular receptor. The hormone–receptor complex binds to specific DNA sequences and acts as a transcription factor, switching on the transcription of particular genes, for example in cells of the uterus lining (C2.1).
A biochemical: lactose in E. coli
The genes for enzymes that take up and digest lactose are grouped together in the lac operon. When lactose is absent, a repressor protein binds to the DNA next to the promoter, blocking RNA polymerase, so the genes are not transcribed. When lactose is present, a form of it binds to the repressor, changing its shape so it detaches from the DNA. RNA polymerase can now transcribe the genes, and the enzymes are made.

The lac operon makes biological sense: the bacterium makes lactose-digesting enzymes only when lactose is available, not wasting energy and amino acids on enzymes it cannot use.

✏️Worked example

(a) In E. coli growing in a medium with glucose but no lactose, predict whether the enzymes for lactose digestion are produced, and explain. Then predict what happens when lactose is added and glucose is used up.
(b) A mutation changes the repressor so that it can no longer bind to DNA. Predict the effect.
(c) A particular mRNA has a half-life of 2 hours in a cell (half of the molecules present are degraded every 2 hours). If transcription stops when 8000 molecules are present, calculate how many remain after 6 hours, and explain why controlling mRNA degradation regulates the amount of protein made.

(a) With no lactose, the repressor is bound to the DNA next to the promoter of the lac operon, blocking RNA polymerase, so the genes are not transcribed and the enzymes are not produced. When lactose is added, it binds to the repressor, changing its shape so that the repressor detaches from the DNA. RNA polymerase binds, the genes are transcribed and translated, and the enzymes are made, allowing lactose to be taken up and digested.

(b) The repressor could never block transcription, so the enzymes would be produced all the time, whether or not lactose was present. This wastes energy and amino acids when lactose is absent.

(c) 6 hours is three half-lives:

\[ 8000 \times \left( \tfrac{1}{2} \right)^{3} = 8000 \times \tfrac{1}{8} = 1000\ \text{molecules} \]

The rate of translation depends on how many mRNA molecules are available. An mRNA that is degraded quickly is translated for only a short time, so production of its protein stops soon after transcription stops; an mRNA that persists keeps being translated for hours or days. By changing how quickly nucleases break down an mRNA, a cell can control how much protein is made, and how quickly production responds to change.

Check it. Halve step by step: 8000 → 4000 (2 h) → 2000 (4 h) → 1000 (6 h). If your answer is 8000 ÷ 6 or 8000 − 3 × 4000, you have treated decay as linear rather than halving.
Saying lactose “switches on the gene” or “binds to the DNA”. Lactose does not interact with DNA at all. It binds to the repressor, which then releases the DNA. The gene is expressed because a block has been removed, not because lactose activates it directly. Name the repressor in every answer.

📝Practise

Work through these on paper, then reveal the answer. All are HL only.

1. Distinguish between the genome, the transcriptome and the proteome of a cell.
The genome is all the genetic information of the organism — its complete DNA sequence, which is the same in almost all its cells. The transcriptome is all the RNA transcribed in a particular cell at a particular time. The proteome is all the proteins produced by the cell at that time. Because no cell expresses all its genes, the transcriptome and proteome differ between cell types and change with time and conditions, while the genome does not.
2. Explain how enhancers and transcription factors regulate transcription.
Transcription factors are proteins that bind to specific base sequences in DNA. Some bind to the promoter, helping RNA polymerase to bind and start transcription. Enhancers are DNA sequences, sometimes far from the gene, to which activator transcription factors bind; the DNA loops so that the bound activators interact with the promoter complex, increasing the rate of transcription. Different cells contain different transcription factors, so different genes are transcribed.
3. Explain how methylation of DNA can silence a gene.
Methyl groups are added to cytosine bases in the DNA of the gene’s promoter. Heavy methylation prevents transcription factors and RNA polymerase from binding to the promoter (and can attract proteins that pack the DNA tightly). The gene is therefore not transcribed, no mRNA is made, and the gene is not expressed. The base sequence itself is unchanged.
4. Explain what is meant by epigenetic inheritance.
Epigenetic inheritance is the passing of a phenotypic change to daughter cells or offspring without any change in the nucleotide sequence of DNA. It happens when epigenetic tags, such as DNA methylation or histone modification, remain in place through mitosis (so a pattern of gene expression is kept in a cell lineage) or through meiosis (so it is passed to the next generation). The altered pattern of gene expression, not the genes themselves, is inherited.
5. Explain how studies of monozygotic twins provide evidence that the environment affects gene expression.
Monozygotic twins develop from the same zygote, so they have identical genomes. Any differences in their phenotype cannot be due to differences in DNA sequence and must be due to the environment. Studies show that twins’ patterns of DNA methylation (epigenetic tags) are very similar early in life but become more different as they age, especially if they live apart or have different lifestyles. This indicates that environmental factors alter epigenetic tags and so gene expression.
6. A liger is much larger than both of its parents, but a tigon is not. Explain this difference.
Ligers and tigons receive the same two sets of genes but from opposite parents. Most epigenetic tags are removed in gametes, but not all: some growth-related genes are imprinted, so only one parent’s copy is expressed. In lions, paternal genes promote growth strongly, and maternal lion genes restrain it. A liger has a lion father (strong growth promotion) and a tiger mother, whose genes have not evolved to counteract it, so growth is unrestrained and it becomes very large. A tigon has a lion mother (growth-restraining imprinting) and a tiger father, so it is not enlarged. The difference is epigenetic, not a difference in genes.

🔗Go deeper — other people’s work

These are external resources, not mine. If one stops working, tell me and everything above it on this page still stands.

  • Learn.Genetics (University of Utah) — the Epigenetics module, including identical twins and imprinting.
  • HHMI BioInteractive — animation of the lac operon.
  • Nature Education, Scitable — articles on transcription factors, enhancers and DNA methylation.